5 months of the original injury. It had been postulated that since HBOT stimulates the activity of osteoblasts and decreases development of fibrosis [fifty four], it interrupts The fundamental vicious cycle of CRPS pathophysiology., contributors undergoing HBOT shown an increase in nerve conduction velocities. This suggests which the therapy may fa
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Greater mechanical withdrawal threshold, prolonged thermal withdrawal latency, reduced nNOS and iNOSAngiogenesis: The therapy promotes the formation of latest blood vessels, enhancing circulation in influenced regions. This angiogenic impact is especially useful since it helps restore blood supply to nerves which were deprived of adequate oxygen an
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